現在因癌症死亡的人數終於超過了心血管疾病。經常看到周遭的朋友患癌而去,新聞上看到名人富人白人黑人都不能幸免。大家真是談癌色變。我的母親也是因癌症而去世的。想到得癌症後的開刀,放療,化療所帶來生不如死的痛苦,其恐懼感不可想象。
隨著科學家們對於癌症的不斷研究,最近終於從分子生物學的角度對於癌症的生長過程有了突破性的發現。美國哈佛大學醫學院發現了控製癌細胞生長的分子開關。如果對於該分子開關進行控製,那麽癌細胞可以轉變成正常細胞。
盡管當今癌症理論學派無數,但歸根到底癌症是個代謝病。糖尿病是個代謝病,而癌症實際是個另類糖尿病。糖尿病與癌症同屬代謝病,是兩個不同的極端。從細胞層次上講,糖尿病是細胞失去了使用糖的能力,由於缺乏能量而慢慢退化,最後導致器官衰竭。癌症是細胞無 節製地吸入糖導致細胞無節製生長,最後導致破壞器官的正常功能。對於糖尿病病人得癌症後死亡率很高。好消息是癌症和糖尿病的治療方法基本相同,因為同為代 謝病,其平衡點是一樣的。
抗癌總願則:少吃米麵,多吃素菜,適量葷食。達到和保持弱堿體質。保持血液幹淨,營養充足。跟據最新研究發現,四十歲以上者體內都有微型癌腫瘤,在大量補充抗氧劑的情況下,微型癌腫瘤不會長大,最後會消失。在腫瘤消失時,其大小便會有強烈的臭味。
我將這幾年幫助他人調理的體會整理了一下同大家分享和討論。
中文講座的連接為,
http://www.tudou.com/programs/view/0OVMVM793gI/
英文講座的連接為,
http://www.tudou.com/programs/view/p2vVRAtRMWQ/
聲明,我不是醫生,這個講座是教育性質的。有病看醫生噢。
參考資料,
http://www.sciencemag.org/cgi/content/abstract/324/5930/1029
Understanding the Warburg Effect: The Metabolic Requirements of Cell Proliferation
Matthew G. Vander Heiden,1,2 Lewis C. Cantley,2,* Craig B. Thompson3,*
In contrast to normal differentiated cells, which rely primarily on mitochondrial oxidative phosphorylation to generate the energy needed for cellular processes, most cancer cells instead rely on aerobic glycolysis, a phenomenon termed "the Warburg effect." Aerobic glycolysis is an inefficient way to generate adenosine 5'-triphosphate (ATP), however, and the advantage it confers to cancer cells has been unclear. Here we propose that the metabolism of cancer cells, and indeed all proliferating cells, is adapted to facilitate the uptake and incorporation of nutrients into the biomass (e.g., nucleotides, amino acids, and lipids) needed to produce a new cell. Supporting this idea are recent studies showing that (i) several signaling pathways implicated in cell proliferation also regulate metabolic pathways that incorporate nutrients into biomass; and that (ii) certain cancer-associated mutations enable cancer cells to acquire and metabolize nutrients in a manner conducive to proliferation rather than efficient ATP production. A better understanding of the mechanistic links between cellular metabolism and growth control may ultimately lead to better treatments for human cancer.
1 Department of Medical Oncology, Dana-Farber Cancer Institute, Boston, MA 02115, USA.
2 Beth-Israel Deaconess Cancer Center and Department of Systems Biology, Harvard Medical School, Boston, MA 02115, USA.
3 Department of Cancer Biology, Abramson Cancer Center, University of Pennsylvania, Philadelphia, PA 19104 USA.
http://www.sciencedaily.com/releases/2010/01/100122222228.htm
Potential Way to Reverse Cancer Cell Metabolism and Tumor Growth
ScienceDaily (Jan. 25, 2010) — A team of scientists led by Professor Adrian Krainer, Ph.D., of Cold Spring Harbor Laboratory has discovered molecular factors in cancer cells that boost the production of an enzyme that helps alter the cells' glucose metabolism. The altered metabolic state, called the Warburg effect, promotes extremely rapid cell proliferation and tumor growth.
http://www.sciencedaily.com/releases/2008/03/080312141243.htm
Fundamental Process Of Tumor Growth Explained
ScienceDaily (Mar. 13, 2008) — Nearly 80 years ago, scientist Otto Warburg observed that cancer cells perform energy metabolism in a way that is different from normal adult cells. Many decades later, this observation was exploited by clinicians to better visualize tumors using PET (positron emission technology) imaging. But it has not been known exactly how tumor cells perform this alternate metabolic feat, nor was it known if this process was essential for tumor growth.